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Carcinogenesis

  • All cells have mechanisms regulating their growth, differentiation and death
  • Cancer develops when cells escape from the normal control mechanism
  • Proliferation is uncontrolled
  • Cells develop ability to invade and metastasise
  • Carcinogenesis is a multi-step process
    • Cells accumulate succession of gene mutations
    • Each mutation overcomes natural anti-cancer defence mechanisms
    • Growth regulation is lost
  • Most cancers result from series of genetic errors

Cancer genes

  • Genes related to cancer development may be divided into
    • Oncogenes
    • Tumour suppressor genes
  • In health, activity of these genes is closely regulated
  • Allows differentiated growth of normal tissues
  • In cancer, balanced control of growth is lost

Oncogenes

  • Oncogenes are regulatory genes
  • Activity is abnormally increased after a genetic alteration
  • Oncogene activation may occur after
    • Chromosomal translocation
    • Gene amplification
    • Mutation within coding sequence of oncogene
  • Oncogenes act in a dominant fashion
  • Examples of oncogenes include:
    • ras on chromosome 11 - mediates signal transduction
    • erbB2 on chromosome 7 - growth factor receptor
    • src on chromosome 20 - tyrosine kinase
    • myc on chromosome 8 - transcription factor

Tumour suppressor genes

  • Code for inhibitory proteins
  • Normal function is to prevent cell growth
  • In cancer, suppressor function is lost
  • Most tumour suppressor genes are recessive
  • Inactivation of tumour suppressor genes can occur by
    • Gene mutation causing loss of gene product
    • Prevention of binding of a gene product to its target site
    • Inactivation by other proteins
  • Examples of tumour suppressor genes include
    • Rb on chromosome 13 - control of cell cycle
    • p53 on chromosome 17 - DNA repair and apoptosis
    • Bcl2 on chromosome 18 - apoptosis
    • APC on chromosome 5 - regulation of co-transcriptional activators
  • Mutation of tumour suppressor genes is seen in many familial cancers
    • Rb - childhood retinoblastoma
    • p53 - Li-Fraumeni syndrome
    • APC - familial colon cancer
    • BRCA1/2 - familial breast cancer

Cell growth

  • In health growth factors are made by one cell type to stimulate another
  • Malignant cells generate their own stimulatory growth factors
  • Positive feedback cycle occurs by two mechanisms
    • Alteration to growth factors or receptors
    • Alteration to intracellular signalling pathways

 

Author:  Dr Shakeeb Khan

 

 
 

Last updated: 05 January 2008

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